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Internal Validity

Internal validity refers to the accuracy of statements made about the causal relationship between two variables, namely, the manipulated (treatment or independent) variable and the measured variable (dependent). Internal validity claims are not based on the labels a researcher attaches to variables or how they are described but, rather, to the procedures and operations used to conduct a research study, including the choice of design and measurement of variables. Consequently, internal validity is relevant to the topic of research methods. In the next three sections, the procedures that support causal inferences are introduced, the threats to internal validity are outlined, and methods to follow to increase the internal validity of a research investigation are described.

Causal Relationships between Variables

When two variables are correlated or found to covary, it is reasonable to ask the question of whether there is a direction in the relationship. Determining whether there is a causal relationship between the variables is often done by knowing the time sequence of the variables; that is, whether one variable occurred first followed by the other variable. In randomized experiments, where participants are randomly assigned to treatment conditions or groups, knowledge of the time sequence is often straightforward because the treatment variable (independent) is manipulated before the measurement of the outcome variable (dependent). Even in quasi-experiments, where participants are not randomly assigned to treatment groups, the investigator can usually relate some of the change in pre-post test measures to group membership. However, in observational studies where variables are not being manipulated, the time sequence is difficult, if not impossible, to disentangle.

One might think that knowing the time sequence of variables is often sufficient for ascertaining internal validity. Unfortunately, time sequence is not the only important aspect to consider. Internal validity is also largely about ensuring that the causal relationship between two variables is direct and not mitigated by a third variable. A third, uncontrolled, variable can function to make the relationship between the two other variables appear stronger or weaker than it is in real life. For example, imagine that an investigator decides to investigate the relationship between class size (treatment variable) and academic achievement (outcome variable). The investigator recruits school classes that are considered large (with more than 20 students) and classes that are considered small (with fewer than 20 students). The investigator then collects information on students’ academic achievement at the end of the year to determine whether a student's achievement depends on whether he or she is in a large or small class. Unbeknownst to the investigator, however, students who are selected to small classes are those who have had behavioral problems in the previous year. In contrast, students assigned to large classes are those who have not had behavioral problems in the previous year. In other words, class size is related negatively to behavioral problems. Consequently, students assigned to smaller classes will be more disruptive during classroom instruction and will plausibly learn less than those assigned to larger classes. In the course of data analysis, if the investigator were to discover a significant relationship between class size and academic achievement, one could argue that this relationship is not direct. The investigator's discovery is a false positive finding. The relationship between class size and academic achievement is not direct because the students associated with classes of different sizes are not equivalent on a key variable—attentive behavior. Thus, it might not be that larger class sizes have a positive influence on academic achievement but, rather, that larger classes have a selection of students that, without behavioral problems, can attend to classroom instruction.

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