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The causes of chronically elevated blood pressure, formally known as hypertension (HT), are not well understood by medical scientists and practitioners although there has been a long-standing belief that mental or emotional factors may be involved. During the 1930s-1950s, psychoanalytically oriented practitioners and researchers, representing the psychosomatic movement, emphasized that mental and emotional distress can contribute to the development of physical disorders. Two leaders of the psychosomatic movement, Flanders Dunbar and Franz Alexander, proposed that inhibition of angry feelings contributes to the development of HT. They reasoned that hostile provocation typically is associated with increases in blood pressure (BP), but after the anger is expressed, BP typically decreases. However, according to the anger-suppression hypothesis, some persons are personally predisposed to suppress their rage. This leads to recurrent autonomic arousal, which eventually leads to chronically elevated BP. According to this idea, the habitual tendency to suppress angry feelings was conceptualized as a personality trait that represented a fear or anxiety about expressing anger to others. Alexander also thought that some constitutional physical predisposition and a stressful, precipitating condition, such as a serious interpersonal conflict or death of a close relative, were also necessary conditions for the development of HT, but these qualifiers have not tended to receive attention from subsequent researchers.

From its inception, the idea of a “hypertensive personality” was controversial among physicians who were skeptical about a role for emotional and mental factors in the development of HT. Although the psychoanalytic approach waned in influence, interest in this topic reemerged with the rise of behavioral medicine and health psychology. In the past three decades, nearly 50 studies testing the anger expression hypothesis have been published.

Cross-Sectional Evidence

Most studies testing this idea have been crosssectional in design. In such studies, HTs were identified at medical clinics as a result of self-referral and constituted the “cases” whose responses on the anger scales are compared with responses of nonpatients unconnected to the medical clinic. Other studies also have been cross-sectional but sampled from larger populations and identified HTs through community screening.

Studies have used a variety of self-report measures to assess the individual's habitual tendency to express anger, but only a few scales have adequate validation. Charles Spielberger and Ernest Harburg developed the most popular instruments to measure personal tendencies to express (anger-out) and suppress (anger-in) anger. A general problem in this literature, however, is that there is little consensus about the best way to measure anger expression because anger is a multidimensional and thus potentially ambiguous construct. In addition, although these scales purport to assess personal reluctance to express annoyance and irritation, they also are strongly saturated with more general negative feelings. Consequently, it may not be anger-in per se that is responsible when positive results are obtained.

Narrative and quantitative reviews of the empirical literature indicate that several studies report positive associations, typically of modest to moderate strength, between anger suppression and BP, consistent with the psychosomatic hypothesis. However, the results across studies are highly variable. One recent study employing an extensive series of psychological measures, including anger expression, and assessing BP in the clinic and using an ambulatory BP monitor for 24 hours during a normal workday failed to find any significant associations between personality and BP.

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